Oversimplifying, spinal cord stimulation creates tiny currents that stop the injured nerves’ aberrant firing, acting somewhat like a heart pacemaker. Generally speaking, the human brain can filter out incoming information. As a result, the brain prioritizes environmental information, perceptions, and sensations required for survival and personal well-being. Under normal circumstances, pain is a human defense mechanism, a warning sign of impending bodily harm. But, after nerve damage, pain loses its protective functions and becomes. Chronic nerve pain from diabetic peripheral neuropathy and sciatic nerve pain is caused by damaged nerve fibers that mislead the brain. Small electrodes are placed around the spinal cord injured nerve fibers during spinal cord stimulation, stopping or preventing erroneous pain signals from reaching the brain. The spinal cord prioritizes non-pain nerve fibers, interrupting pain signal transmission and alleviating chronic nerve pain. In a nutshell, Once the electrodes are placed, and the spinal cord is stimulated, the damaged nerve fiber pain signal won’t reach the brain. In other words, the pain perception from the damaged nerve fiber will significantly decrease. To clarify, stimulation doesn’t eliminate the pain. It modifies the brain’s perception of the pain.